肺心病血液光量子治疗前后血液流变性质变化及机制探讨  被引量:3

HEMORHEOLOGICAL CHANGES AND POSSIBLE MECHAMSM IN PATIENTS WITH PULMONARY HRART DISEASE BEFORE AND AFTER ULTRA VIOLET-RADIATION TREATMENT

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作  者:张周良[1] 刘茂贤[1] 李秋生[1] 邓明亮[1] 彭长福[2] 

机构地区:[1]第四军医大学唐都医院检验科,陕西西安710038 [2]第四军医大学唐都医院呼吸科,陕西西安710038

出  处:《中国血液流变学杂志》1992年第2期5-8,共4页Chinese Journal of Hemorheology

摘  要:15例肺心病血液高粘滞患者自体血250ml用XZY-Ⅰ型量子血液治疗机进行紫外线照射和充以纯氧后再输入,每周一次,最多连续做三次(平均1.86±0.71次)后,作者发现血液全血高切、低切粘度,全血高切、低切还原粘度,血浆比粘度,血浆纤维否白原含量,红细胞聚集指数,红细胞电泳率等有显著性改变,单纯常规治疗组15例患者血液流变无此变化,分析认为,这些变化可能与红细胞聚集性降低、纤维蛋白源溶解度提高及血氧饱和度增加有关。patients with pulmonar heart disease and with high blood viscosity syndrome underwent auto -blood (250ml) retransfusion.Before retransfusion the blood was ultraviolet - radtated and oxyge - charged with XZY-I Quantum Blood Therapeutic Instrument. The performance was employed once a week, or at most trice a week (mean 1.86±0.71times). Patients' whole blood vicosity (in high and low shear), plasma specific viscosity, whole blood reductive viscosity, (in high and low shear), RBC aggregation index, fibrinogen, electrophoretic rate and Casson stress are changed obviously.(p <0.01, P<0. 05);the blood viscosity of the group (15 patients) treated by routine revealed no change. Causes of these changes may be the direct decreasing of cells aggregation, ascending of fib- rinogen solubility and increasing of the oxygenated hemoglobin satuation.

关 键 词:肺心病 光量子治疗 血液流变学 

分 类 号:R4[医药卫生—临床医学]

 

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