在肠系膜上动脉闭塞性休克发生中循环血量和血液流变学变化  

Effect of the Changes in the Blood Volume and Hemorhelogy during Superior Mesentric Artery Occlusion Shock

作　　者：李化民[1] 吴中立[2] 张金山[2] 

机构地区：[1]锦州医学院病理生理学教研室,锦州121004 [2]第二军医大学病理生理学教研室

出　　处：《基础医学与临床》1993年第4期43-45,共3页Basic and Clinical Medicine

摘　　要：夹闭成年大耳白兔肠系膜上动脉,以复制SMAO休克动物模型。休克后,颈动脉血压下降,循环血量减少,血细胞压积升高,血小板聚集率下降;血压下降与循环血量减少密切相关(r=0.6958,P<0.05)。提示:血液浓缩、循环血量减少,是引起SMAO休克的一个重要因素。The superior mesentric artery occlusion (SMAO)shock model was induced by clamping the superior mesentric artery (SMA) in the adult rabbits. After shock, the blood pressure of the cervical artery and the blood volume decreasedjwhile the hemo-tocrit and the blood viscosity increased) significantly. The decrease of blood pressure was significantly related with the reduction of blood volume (r = 0.6958, P<0.05) .This results shows that the blood concentration and the decrease of the blood volume plays an important part in inducing SMAO shock.

关 键 词：循环血量 休克 血液流变学 

分 类 号：R441.9[医药卫生—诊断学]