次声生物学效应的相关机制  

The underlying mechanisms for infrasonic bioeffects

作　　者：庄志强[1] 裴兆辉[2] 陈景藻[2] 

机构地区：[1]第四军医大学预防医学系放射医学教研室,陕西西安710032 [2]西京医院康复与理疗科,陕西西安710032

出　　处：《疾病控制杂志》2005年第4期328-330,共3页Chinese Journal of Disease Control and Prevention

基　　金：国家自然科学基金资助项目(30400091)

摘　　要：次声广泛存在于环境中,达到一定强度时可以产生某些生物学效应,其基本机制是产生生物共振并引起理化效应,进一步导致机体自由基增多、脑组织谷氨酸浓度和代谢型谷氨酸受体(mGluR)变化、钙超载以及血管内皮内分泌功能改变等。本文综述了次声生物学效应的相关机制及特点,为本领域的研究提供理论参考。Infrasound exists widely in circumstances, which will lead to some bioeffects if its intensity is high enough. The basic mechanism is the biological resonance causing physical changes and chemical reactions, which result in the increase of free radicals, changes of the glutamate concentration and the receptors of metabotropic glutamate in the tissues of brain, calcium overload and changes on the function of endothelial endocrine subsequently. This article summarized the underlying mechanisrns and characters of infrasonic bioeffects to provide theoretical references for this field.

关 键 词：声刺激 自由基 受体 谷氨酸 

分 类 号：R852.24[医药卫生—航空、航天与航海医学] R340.4[医药卫生—临床医学]