失重或模拟失重条件下血管收缩反应变化的研究进展  

Progress in Research of Vascular Contractility under Weightlessness or Simulated Weightlessness.

作　　者：李志利[1] 姜世忠[1] 

机构地区：[1]航天医学工程研究所,北京100094

出　　处：《航天医学与医学工程》2005年第5期387-390,共4页Space Medicine & Medical Engineering

基　　金：中国载人航天工程资助项目

摘　　要：失重或模拟失重条件下血管收缩功能的改变是立位耐力下降的重要原因。血管功能的实现与血管内的压力、血流状态和血管结构有密切关系,由于血管所处部位不同,其结构和特点也不尽一致。失重或模拟失重时流体静压消失和血液的头向分布,使身体各部位血管的结构和功能发生着不同的变化,本文就近年来有关这方面的研究作了简要的回顾。Weightlessness or simulated weightlessness-induced orthostatic intolerance appears to be due, in part, to the change of vascular contractility. The functions of vessels are close to blood pressure, vessels structure and blood flow. The vessels have respective structural and functional characteristics due to their different anatomic regions. The cephalic fluid shift and elimination of the head-to-foot hydrostatic pressure gradient in weightlessness or simulated weightlessness may in- duce adaptations of the vessels, which are different in property and degree. This review summarizes recent researches about these adaptations to weightlessness or simulated weightlessness.

关 键 词：失重模拟 立位耐力 血管 收缩 

分 类 号：R852.22[医药卫生—航空、航天与航海医学]