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作 者:陆建学[1] 王建钢[1] 周凯[1] 盛文权[1] 夏连军[1] 黄宁宇[1] 施兆鸿[1] 赵荣兴[1]
机构地区:[1]中国水产科学研究院东海水产研究所农业部海洋与河口渔业重点开放试验室,上海200090
出 处:《海洋渔业》2006年第1期55-59,共5页Marine Fisheries
基 金:中国水产科学研究院科研基金项目(2001-1-1);上海市科学技术委员会资助项目(043919348)
摘 要:在海水和半咸水中,用高锰酸钾(KMnO4)对黑鲷幼鱼进行急性毒性试验。结果表明:在海水盐度27.3和半咸水盐度10.4条件下,高锰酸钾对黑鲷幼鱼24h的LC50分别为3.41mg/L和4.78mg/L;最高安全使用剂量分别为1.12mg/L和1.68mg/L。半咸水条件下,高锰酸钾对黑鲷幼鱼的急性毒性相对较低,因此,高锰酸钾对黑鲷幼鱼进行药物处理比在海水条件下更安全。高锰酸钾引起鱼体组织发生病理变化是暂时的,一旦受试鱼转到无高锰酸钾的清水环境中,鱼体就会恢复正常。In this paper, the acute toxicity of potassium permanganate to black seabream Sparus macrocephalus juveniles in seawater and brackish water was studied. The results showed that the LC50 values of potassium permanganate in 24h were 3.41 in brackish water ( salinity 10.4‰) and 4.78 mg/L in seawater ( salinity 27.3‰), respectively. The safe doses of potassium permanganate in brackish water and in seawater were 1.12mg/L and 1.68mg/L, respectively. In brackish water, the acute toxicity of potassium permanganate to Sparus macrocephalus juveniles was lower than that in seawater. Therefore, it was much safer to have Sparus macrocephalus juveniles treated in brackish water. The results also indicated that black seabream would return to the normal condition once it was replaced to fresh brackish water or seawater, because the pathological changes caused by potassium permanganate were temporary.
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