穴位离子导入对大鼠心脏超微结构及功能的影响  

Effects of Acupoint Iontophoresis on Ultrastructure and Function of Heart of Rats

作　　者：杨翼[1] 李章华[2] 何金森[3] 

机构地区：[1]武汉体育学院运动医学教研室,湖北武汉市430079 [2]武汉大学人民医院骨科,湖北武汉市430030 [3]上海中医药大学针推学院,上海市200032

出　　处：《中国康复理论与实践》2006年第2期126-128,F0003,共4页Chinese Journal of Rehabilitation Theory and Practice

基　　金：湖北省教育厅科研重点项目(D200533002)。

摘　　要：目的观察穴位离子导入对大鼠心脏功能的保护机制。方法随机将46只雄性SD大鼠分成5组,采用组织学、透射电镜及放射免疫法观察穴位离子导入对大鼠心脏超微结构的影响。结果穴位离子导入能显著延长大鼠力竭运动时间,保护心肌超微结构,防止心脏异常增大,而且能有效改善因过度训练引起的内皮素和降钙素基因相关肽分泌失衡,减少过度训练对心脏功能的损害。结论心源性运动性疲劳的发生与多因素相关;穴位离子导入能多环节、多层面地保护心肌和改善心脏功能,从而延缓疲劳产生和促进疲劳消除。Objective To research the protoctive mechanism of acupoint iontophoresis to the heart function. Methods 46 SD rats were randomly divided into 5 groups. The methods of histology, transmission electron microscope and radioimmunoassay were used respectively to observe the influence of acupoint iontophoresis on rats＇ heart uhrastructure. Results Acupoint iontophoresis could prolong rats＇ exertion period, protect uhrastructure of heart and prevent it from abnormal accretion. Over training would cause unbalance secreting between endothelin （ET） and calcitoningene related peptidc （CGRP）, while acupoint iontophoresis could improve such situation. Conclusion The rise of exercise induced fatigue is related to many factors; acupoint iontophoresis can protect cardiac muscle, improve heart function and prevent it from exercise induced fatigue via several ways.

关 键 词：运动性疲劳 穴位离子导入 心脏功能 大鼠 

分 类 号：R454.1[医药卫生—治疗学]