快速性恶性室性心律失常的发作方式及其临床干预  被引量:8

Initiating mode and clinical intervention of malignant ventricular tachycardia

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作  者:陈增瑞[1] 徐金女 陈美霞[1] 黄锡通[1] 

机构地区:[1]浙江省玉环县人民医院ICU,317600

出  处:《心电学杂志》2006年第2期80-81,84,共3页Journal of Electrocardiology(China)

摘  要:目的探讨快速性恶性室性心律失常的发作方式及其临床干预。方法对我院收治的快速性恶性室性心律失常56例(入院后至少发生1次以上),根据基础Q-T间期是否延长分为Q-T间期正常和长Q-T(U)间期,并按不同的发作方式临床干预治疗。结果基础心律Q-T间期正常(0.38±0.03s)33例,以室性心动过速或多形性室性心动过速多见,多数可用直流电复律。而Q-T间期延长(0.51±0.04s)23例,以尖端扭转型室性心动过速多见,多数经大剂量补钾、镁+异丙肾上腺素或阿托品静脉治疗。结论快速性恶性室性心律失常由不同形式的室性期前收缩诱发,应针对不同的临床、心电学特征,采取临床干预措施。Objective To investigate initiating mode and clinical intervention of malignant ventricular tachycardia. Methods 56 cases with malignant ventricular tachycardia were divided into normal Q-T interval group(33 cases) or long Q-T(U) interval group(23 cases) based on basic Q-T interval. Initiating mode of ventricular tachycardia and it's clinical intervention were analyzed and compared between two groups. Results In normal Q-T interval group, most of patients had polymorphic ventricular tachycardia attacking and received electrical conversion. In long Q-T interval group, torsade de pointes was main pattern of ventricular tachycardia and large doses of potassium and magnesium plus isoprenaline or atropine were given intravenously in most patients. Conclusion Malignant ventricular tachycardias have different electrocardiographic characteristics which need different clinical intervention.

关 键 词:室性期前收缩 恶性心律失常 发作方式 干预 

分 类 号:R541.7[医药卫生—心血管疾病]

 

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