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作 者:邹宇炜[1] 张秀琴[2] 杨新明[1] 苏峰[1] 潘进社
机构地区:[1]河北北方学院附属第一医院骨科,河北张家口075000 [2]暨南大学医学院口腔医学系,广东广州510632 [3]河北省第三人民医院骨科,河北石家庄050000
出 处:《河北北方学院学报(医学版)》2006年第5期1-3,共3页Journal of Hebei North University:Medical Edition
摘 要:目的:探讨外踝短缩所致创伤性踝关节炎发生的生物力学机制。方法:选取8只新鲜冷冻尸体下肢,分别在外踝正常长度、短缩1/3、短缩2/3和短缩3/3的情况下,测量和分析踝关节面应力分布的变化。结果:踝关节应力接触区大致呈三角形,外踝短缩后踝关节面应力分布发生变化,以向前、内、外侧压力增高最为显著。结论:外踝短缩后踝关节面应力分布发生明显变化,这可能是导致创伤性关节炎发生的重要机制。Objective: To investigate the biomechanism of traumatic ankle arthritis resulted from shortened lateral malleolus. Methods;The lower extremity specimens of eight fresh frozen cadaver were used. At the lengthes of normal,shortened 1/3,shortened 2/3 and shortened 3/3 lengthes, the tibial astragaloid joint's area and pressure were measured. Results: The contact area of the normal tibial astragaloid joint looked like triangular analyzed by shapes of dyeing area. The stress force of the tibial astragaloid joint changed in the consequence of shortened lateral malleolus, anti it increased especially to the front, inside and lateral. Conclusion:The stress force of the tibial astragaloid joint will changed in the consequence of shortened lateral malleolus, maybe it is the important mechanism of traumatic ankle arthritis.
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