一氧化氮与肺纤维化的研究进展  

Research progress on nitric oxide and pulmonary fibrosis

作　　者：孙玉敏[1] 宋福成[2] 陈晓玲[3] 

机构地区：[1]沧州医学高等专科学校内科教研室,沧州061001 [2]武警8642部队卫生队,定州072353 [3]河北医科大学病理生理研究室,石家庄050017

出　　处：《现代生物医学进展》2006年第9期105-106,118,共3页Progress in Modern Biomedicine

摘　　要：肺纤维化是一组由多种因素引起的肺间质性病变,肺纤维化的发病机制迄今尚未完全清楚。近年来,发现在哺乳动物细胞的一氧化氮合酶催化合成的一氧化氮在肺纤维化的发生发展中发挥着重要的作用。因此,阐述一氧化氮与肺纤维化的关系,有着重要的理论意义和潜在的临床应用价值。Pulmonary fibrosis is a set of pulmonary interstitial pathological changes caused by many factors. Its mechanisms are not fully understood. In recent years, some scholars discover that in mammal cells nitric oxide , catalyzed and synthesized by nitric oxide synthase, plays an important role in pulmonary fibrosis. So it is of important academic significance and potential clinical value to expound the relationship between nitric oxide and pulmonary fibrosis.

关 键 词：一氧化氮 一氧化氮合酶 肺纤维化 

分 类 号：R563[医药卫生—呼吸系统]