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机构地区:[1]解放军总医院老年心血管二科,北京100853 [2]河北医科大学第三医院超声科
出 处:《中华超声影像学杂志》2007年第2期104-107,共4页Chinese Journal of Ultrasonography
摘 要:目的探讨左室壁不同部位局部心肌肥厚的发生率及左室假腱索与局部心肌肥厚的关系。方法应用二维超声测量52例局部心肌肥厚患者各节段室壁厚度、最厚及最薄室壁节段数目、增厚室壁节段数目,统计分析最厚及增厚发生的部位以及不同室壁厚度情况下假腱索附着点阳性率的差异。结果局部心肌肥厚组最厚节段发生率最高的部位是室间隔基部,其次为左室侧壁心尖段;室间隔基部及左室侧壁心尖段的假腱索附着点阳性率显著高于左室其他室壁节段。局部心肌肥厚组增厚节段假腱索附着点的阳性率与非增厚部位假腱索附着点的阳性率相比差异有统计学意义(54/71对66/761,χ^2=233.47,P〈0.0001);局部心肌肥厚组增厚节段假腱索附着点的阳性率(76.1%)与最薄节段假腱索附着点的阳性率(2.5%)相比较差异有统计学意义(χ^2=84.7,P〈0.001);局部心肌肥厚增厚节段假腱索附着点的阳性率与最厚节段假腱索附着点的阳性率(94.3%)相比差异有统计学意义(χ^2=8.4,P=0.04)。结论假腱索附着点是局部心肌肥厚的易发部位,假腱索的牵拉可能是局部心肌肥厚始动或触发的综合因素之一。Objective To determine the detectable rate of myocardial hypertrophy in different regions of left ventricular and to find whether there was any relationship between asymmetric myocardial hypertrophy(AMH) and left ventricular false tendons (LVFT). Methods Fifty-two patients with AMH and 50 normal subjects without AMH were studied by echocardiography. The wall thickness of left ventricular, the detectable rates of LVFT in different regions, the positions of LVFT joining to left ventricular wall, and the angles formed by endocardium of intraventricular septum (IVS) and LVFT were statistical analysed in the two groups. Results In AMH group,the most regions of myocardial hypertrophy among left ventricular wall regions were basal IVS and apical lateral wall(all P 〈0.05). The most origins of LVFTs were basal IVS, and the most endings of LVFTs were apical lateral wall(all P G0.05). A significiant difference of the LVFT detectale rate was found between in the basal IVS (or in the apical lateral wall) and in any of the other regions(all P 〈0.05). Also there was a significant difference in the detectable rate of LVFT between LVFT joining to the thickest wall and that joining to the thinnest wall(54/71 vs 2/77,χ^2 = 84.7, P 〈0. 001 ) in AMH group, also between LVFTs joining to the thickest wall and those joining to thicker wall (54/71 vs 50/53,χ^2 = 8.4, P = 0. 04). Conclusions The joint of LVFT to the ventricular wall was the most occurred position of myocardial hypertrophy. The drawing of LVFT at ventricular wall may be one of the compound factors resulting in regional myocardial hypertrophy.
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