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机构地区:[1]聊城市第二人民医院介入科,山东临清252601
出 处:《实用放射学杂志》2007年第5期650-652,664,共4页Journal of Practical Radiology
摘 要:目的探讨青壮年股骨头缺血坏死的发病机理。方法选择经临床和影像检查确诊、并经介入治疗的78例113个缺血坏死股骨头的血管造影资料与45例46个正常股骨头血管造影资料对照分析。结果观察组股骨头供血动脉发育变异28例(35.9%),51支血管,其中髂内动脉壁支变异17支(33.3%),旋股内侧动脉变异29支(56.9%),旋股外侧动脉4支(7.8%),旋股内外侧动脉共干1支(2.0%)。对照组股骨头供血动脉发育异常5例5支,2组比较有显著性差异(P<0.005)。结论股骨头供血动脉发育变异可能是造成股骨头缺血坏死的一个潜在因素。Objective To study the pathogenesis of avascular necrosis of femoral head( ANFH) in young adults. Methods The data of DSA of femoral head (113 femoral heads ) with ANFH proved by clinic or radiology and treated by interventional radiology and 45 normal peoples (46 femoral head ) were comparatively analysed. Results The variation of artery of femoral head in interventional treating group was appeared in 28 cases( 35.9% ) ,51 arteries involved,including 17 internal lilac arteries variation( 33.3% ) ,29 medial femoral circumflex arteries variation (56.9% ) ,4 lateral femoral circumflex arteries variation (7.8% ) , 1 lateral and medial femoral circumflex arteries common bole variation( 2.0% ). The variation of artery of femoral head in control group was appeared in 5 eases,5 arteries involved. There was significant difference between the two groups(P 〈 0. 005 ). Conclusion The variation of the feeding artery of femoral head may be one of potential factors resulted in ANFH.
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