运动性骨骼肌微损伤的特性  被引量:13

Characteristics of exercise-induced micro-damage of skeletal muscle

在线阅读下载全文

作  者:魏源[1] 

机构地区:[1]广州体育学院科学实验中心,广东省广州市510500

出  处:《中国组织工程研究与临床康复》2007年第19期3842-3845,共4页Journal of Clinical Rehabilitative Tissue Engineering Research

基  金:国家科技攻关计划奥运科技专项基金资助(2002BA904B16)~~

摘  要:目的:分析运动性骨骼肌微损伤的特性。方法:应用计算机检索HighWire Press1989-01/2006-12关于运动性骨骼肌微损伤的文章。检索词“musclemicro-damage”并限定文章的语言种类为English。同时利用计算机检索中国期刊全文数据库1989-01/2006-12的相关文章,限定文章语言种类为中文,检索词“运动性骨骼肌微损伤”。结果:运动后骨骼肌微损伤的发生的直接证据主要表现在运动对骨骼肌微结构和超微结构的影响,其间接证据主要表现在血液中肌肉酶活性和肌红蛋白含量增加、血液中C-反应蛋白含量升高、尿液中肌肉蛋白质分解产物增加和骨骼肌组织中炎症反应标志物增高。结论:了解运动性微损伤的特性可以预防和干预运动后微损伤的发生,为有效地预防延迟性肌肉酸痛提供理论参考。AIM: To analyze the features of the exercise induced micro-damage of skeletal muscle. METHODS: A computed-based online search of HighWire Press was undertaken to identify the articles about exercise micro-damage of skeletal muscle published in English between January 1989 and December 2006 with the key words of "muscle micro-damage". Meanwhile, we searched Chinese Journal Full-text Database for related Chinese articles published from January 1989 to December 2006 with the key words of "exercise micro-damage of skeletal muscle" in Chinese. RESULTS: The influence of exercise on the microstructure and ultrastructure of skeletal muscle is the direct evidence for its micro-damage after exercise; the indirect evidence mainly includes the increase in muscular enzyme activity and myoglobin, elevation of blood C-reactive protein, augment of breakdown products of muscle protein in urine and raise of inflammatory reaction markers in skeletal muscle tissues. CONCLUSION: Understanding to the characteristics of micro-damage could prevent and interfere the exercise micro-damage of skeletal muscle, and provide theoretical reference for effective prevention to delayed muscular soreness.

关 键 词:骨骼肌微损伤 延迟性肌肉酸痛 肌肉微细损伤 

分 类 号:R685[医药卫生—骨科学]

 

参考文献:

正在载入数据...

 

二级参考文献:

正在载入数据...

 

耦合文献:

正在载入数据...

 

引证文献:

正在载入数据...

 

二级引证文献:

正在载入数据...

 

同被引文献:

正在载入数据...

 

相关期刊文献:

正在载入数据...

相关的主题
相关的作者对象
相关的机构对象