外源一氧化氮供体对感染轮纹病菌后梨质膜H^+—ATPase及抗氧化酶活性的影响  

Effect of exogenous nitric oxide donor on plasmalemma H^+—ATPase and antioxidative enzyme activities of pear infected by Pnysalosproa piricola Nose.

作　　者：刘招龙[1] 张绍铃[1] 孙益林[1] 乔勇进[1] 

机构地区：[1]南京农业大学园艺学院

出　　处：《中国生态农业学报》2007年第5期105-107,共3页Chinese Journal of Eco-Agriculture

基　　金：国家自然科学基金项目(30170651)资助

摘　　要：梨叶片感染轮纹病菌5d后,用不同浓度的一氧化氮供体硝普钠(SNP)处理,测定了质膜H+—ATPase及抗氧化酶活性的变化。结果表明:浓度为0.3mmol/L SNP处理可提高H+—ATPase、抗坏血酸过氧化物酶(APX)、愈创木酚过氧化物酶(GPX)和谷胱甘肽还原酶(GR)活性,降低脂氧合酶(LOX)活性;浓度为0.1mmol/L和0.6mmol/L SNP处理则对其影响效果不明显;在同一浓度SNP处理组中,随处理时间的延长,其变化不同,较低浓度的SNP可缓解轮纹病菌对梨的伤害作用。Pear leaves were treated by different densities of nitric oxide donor--SNP after its infection with Pnysa- losproa piricola Nose for five days and the activities of plasmalemma H^＋ --ATPase and antioxidative enzyme were investageted. Results show that diseased pear treatment with SNP at the density of 0.3mmol/L can raise H^＋ --ATPase, APX, GPX and GR activities and reduce LOX activity, while 0. l mmol/L and 0.6mmol/L SNP have less apparent effects on pear toxicity mitigation. In the treatment group of SNP at a constant density, there are varying degrees of effect on infected pear with the prolongation of treatment time. SNP in a relatively low density can palliate pear toxicity after Pnyalosproa piricola Nose infection.

关 键 词：NO 梨 轮纹病菌 质膜H^+一ATPaSe抗氧化酶 

分 类 号：S565.4[农业科学—作物学]