肉鸭实验性锰缺乏症及高磷对缺锰影响的病理学研究  

EXPERIMENTAL PATHOLOGY OF MANGANESE (Mn) DEFICIENCY AND EFFECTS OF HIGHER PHOSPHORUS UPON MANGANESE DEFICIENCY IN BROILER DUCKS

作　　者：蔡红[1] 冯泽光[1] 

机构地区：[1]四川农业大学动物科技学院

出　　处：《畜牧兽医学报》1997年第4期342-348,共7页ACTA VETERINARIA ET ZOOTECHNICA SINICA

摘　　要：６９只一日龄天府肉鸭（）随机分为三组，各喂以基础（基础饲粮，含锰１１．６２ｐｐｍ或１０．０５ｐｐｍ）、高磷（基础饲粮＋０．３４％磷）、对照（基础饲粮＋１２０ｐｐｍ锰）饲粮。试验分为两个阶段，第一阶段（０～４周）结束时，每组各留２只鸭继续进行第二阶段（５～２０或２６周）试验。缺锰组第一周开始发病，第二周迅速达到高峰。缺锰病鸭生长明显减缓（ｐ＜０．０１）；多见双腿内弯，站立呈“ｏ”字形；跛行，严重病例以跗关节着地行走；剖检多见双侧跟腱内偏，甚至滑脱；锰浓度分析表明，肝锰含量显著降低（ｐ＜０．０１）。组织学病变可见：胫骨远端骺板增殖区显著增宽（ｐ＜０．０１）；睾丸曲细精管发育障碍。另外。Sixty nine day old Tianfu broiler ducks were randomly divided into 3 groups Group 1 were fed a basal diet (BD) containing 11 62 ppm or 10 05 ppm Mn Group 2 were fed a diet (BD+0 34% phosphorus) with a higher level of phosphorus Group 3 were fed a diet (BD+120 ppm Mn) as a control This experiment was conducted at two stages When the first stage (week 0～4) was ended,2 ducks from every group were reserved to continue the second stage of the experiment (week 5～20 or 26) Group 1 and group 2 appeared clinical symptoms in the first week and got to a peak of illness in the second week The affected ducks showed reduced growth (p<0 01),both legs of“o”shape,lameness,walking with tibia metatarsal joints and both gastrocnemius tendons slipped from their internal condyles Mn levels of livers were decreased significantly (p<0 01) in the Mn deficient groups compared with the Mn supplement group Pathohistological lesions were found by increased width of proliferative zones in epiphyseal growth plates of distal ends of tibias (p<0 01) and prevented development of spermatospores in testes At the same time,ultrastructural changes were observed in epiphyseal growth plates of distal ends of tibias and testes

关 键 词：锰缺乏 高磷 病理学 鸭病 肉用鸭 

分 类 号：S858.326.6[农业科学—临床兽医学]