炎症与急性缺血性肾损伤  

The research progresses of inflammation and acute ischemic kidney injury

作　　者：廖晓辉[1] 张玲[1] 

机构地区：[1]重庆医科大学附属第二医院肾内科,重庆400010

出　　处：《生命的化学》2008年第6期755-758,共4页Chemistry of Life

摘　　要：近年来研究发现细胞间黏附分子-1和单核细胞趋化蛋白-1等炎症因子、核因子-κB及中性粒细胞、单核/巨噬细胞等炎症细胞参与了急性缺血性肾损伤的发生发展,抑制急性缺血性肾损伤时肾脏的炎症反应具有保护肾脏作用。Investigations in recent years have confirmed that intercellular adhesion molecule-l, nuclear factor kappa B, monocyte chemoattractant protein-l, neutrophils, macrophages and lymphocytes were involved in the development of acute ischemic kidney injury （AIKI）. Inhibition of renal inflammation could protect kidney from AIKI.

关 键 词：急性缺血性肾损伤 缺血再灌注 炎症 

分 类 号：R692[医药卫生—泌尿科学]