苏麦3号抗赤霉病性的主基因+多基因混合模型分析  

Major Genes Plus Polygenes Inheritance Analysis of Resistance to Fusarium Head Blight in Sumai 3

作　　者：任丽娟[1] 张旭[1] 周淼平[1] 陆维忠[1] 马鸿翔[1] 

机构地区：[1]江苏省农业科学院农业生物技术研究所,江苏南京210014

出　　处：《江苏农业学报》2008年第6期774-779,共6页Jiangsu Journal of Agricultural Sciences

基　　金：国家"863"项目(2006AA100102);江苏省自然科学基金重点项目(BK2006720)

摘　　要：应用植物数量性状主基因+多基因混合遗传模型对苏麦3号(抗病亲本)与白免3号(感病亲本)杂交组合的R IL群体进行了小麦赤霉病抗性的遗传分析。结果表明,苏麦3号×白免3号重组自交系群体的小麦赤霉病抗性符合3对主基因+多基因控制的加性-上位性模型(G-1模型),主基因遗传率为75%左右,主基因间的互作效应也较显著,而且与主基因的加性效应相反,使赤霉病抗性减弱;赤霉病抗性的多基因效应较大,但其遗传率很小。因此在抗赤霉病育种时不仅要考虑基因间的加性效应,也要注意基因间的互作,同时也要加强微效多基因的效应累加。This study was conducted to elucidate the inheritance of resistance to scab in a recombinant inbred lines （RIL s） population derived from a cross of Sumai 3 （resistant） x Baimian 3 （susceptible） by using the major gene plus polygene mixed inheritance model. Three major genes plus polygene effect （ Model G-1 ） governed the inheritance of resistance to Fusarium head blight in the RILs. The heritability of the major genes was about 75%. The interactive effect between major genes was very significant,which reduced the resistance to scab. Polygene effect was high too, but with low heritability. The additive effect,interactive effect and polygene effect needs to be paid great attention to in wheat scab resistance breeding.

关 键 词：小麦 赤霉病 抗性遗传 主基因+多基因混合遗传模型 

分 类 号：Q348[生物学—遗传学]