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作 者:江滨[1,2] 张寿林[1] 黄金祥[1] 鲁洁波[1] 张金松[1] 宋文佳[1]
机构地区:[1]中国预防医学科学院劳动卫生与职业病研究所 [2]中国医学科学院神经科学研究所
出 处:《中国工业医学杂志》1998年第1期4-7,共4页Chinese Journal of Industrial Medicine
摘 要:目的探讨慢性二硫化碳中毒的早期诊断方法。方法对二硫化碳作业工人进行神经传导速度和脑干听觉诱发电位检测,运用方差分析方法分析检测结果。结果175名二硫化碳作业工人与80例正常人比较,神经传导速度测定15个参数中的13个参数差异有显著意义。对52名二硫化碳作业工人和26名正常人的脑干听觉诱发电位的分析表明,接触组和对照组、不同工龄组别与对照组间脑干听觉诱发电位Ⅰ、Ⅲ、Ⅴ各主波潜伏时和峰间潜伏时未见差异。结论低水平二硫化碳长期接触对听觉神经传导通路影响不大,二硫化碳作用主要靶部位是周围神经。o look for early diagnotic methods of chronic CS2 poisoning,a group of carbon disulfide workers were checked with clinical and electroneurophysiological methods including detection of both nerve conduction velocity(NCVs)and brainstem audiotory evoked potentials(BAEP).The results were analyzed by variance analysisNCV measurement of 175 carbon disulfide workers showed that 13 out of 15 electroneurographic parameters had significant differences compared with the control group(80 healthy people)BAEP detection on 52 carbon disulfide workers and 26 healthy volunteers demonstrated that there were on differences in all the indices of BAEP between these two groups despite of the difference in workingperiodTherefore,the conclusion seems that lowlevel exposure to carbon disulfide only has little effect on the audiotory nerve,but the peripheral nerve may be its target site..
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