外伤性基底节血肿发病机制的探讨  

Pathogenesis of traumatic basis ganglia hematomas

作　　者：刘微波[1] 张新平[1] 

机构地区：[1]四川省科学城医院外二科,四川绵阳621900

出　　处：《西部医学》2009年第5期800-801,共2页Medical Journal of West China

摘　　要：目的探讨外伤性基底节血肿(TBGH)的发病机理。方法对41例外伤性基底节血肿患者,从解剖学基础和受伤时着力情况两方面进行分析,以探讨TBGH的形成机理与治疗结果。结果治愈8例(19.5%),死亡15例(36.6%),其余患者均有不同程度的残废。结论外伤性基底节血肿是由于基底节区特殊的解剖结构,加上特定方向的作用力共同形成。Objective To study the pathogenesis of traumatic basis ganglia hematomas （TBGH）. Methods The clinical data of 41 patients with TBGH were retrospectively analyzed. Results 15 patients died and mortality rate was 36.6%. 8 patients were cured and cure rate was 19. 5%. 18 patients were disabled in various degrees. Conclusion The formation reason of TBGH is the special dissection structure of basis ganglia area.

关 键 词：外伤性基底节血肿 发病机制 着力方向 

分 类 号：R651.15[医药卫生—外科学]