Graves病甲状腺功能亢进症与血糖代谢  

Graves disease hyperthyroidism and glycometabolism

作　　者：孙琦婷[1] 赵德善[1] 

机构地区：[1]山西医科大学第一医院核医学科,太原030001

出　　处：《国际放射医学核医学杂志》2010年第3期143-147,共5页International Journal of Radiation Medicine and Nuclear Medicine

摘　　要：Graves病甲状腺功能亢进症（甲亢）患者常伴有糖代谢紊乱，其机制主要与胰岛β细胞功能受损及胰岛素抵抗有关，而免疫遗传因素也起一定的作用。在^131I和抗甲状腺药物治疗后，伴随着甲状腺激素水平的恢复，血糖逐渐开始下调并恢复正常。因此，对Graves病甲亢伴糖代谢紊乱患者进行积极的早期治疗有助于血糖的控制及并发症的预防。The patients with Graves disease hyperthyroidism are often accompanied by disorder of glycometabolism. β-cell dysfunction and insulin resistance play a main role in this disease,while the immune and genetic factors are also relevant with the disease. The blood glycose level can become normal gradually with the recovery of thyroid hormone after ^131I and antithyroid drug treatment. Therefore, the blood glycose level could be improved and complications could be prevented by early treatment in the patients with Graves disease hyperthyroidism accompanied with glyeometabolism disorder is helpful for improving the blood glycose level.

关 键 词：格雷夫斯病 甲状腺功能亢进症 血糖代谢 碘放射性同位素 近距离放射疗法 

分 类 号：R581.1[医药卫生—内分泌]