获得性长Q-T间期综合征90例临床分析  

Clinical Analysis of Acquired of longQ-T Internal Syndrome

作　　者：孟素芳 

机构地区：[1]河南省郑州市第六人民医院,450000

出　　处：《中国实用医药》2010年第32期17-18,共2页China Practical Medicine

摘　　要：目的分析获得性长Q-T间期综合征的临床特点。方法对90例获得性长Q-T间期综合征病例的临床资料进行回顾性分析。结果 90例中女60例,男30例,发作尖端扭转型室性心动过速1～7/人,平均3.2次,共计29次的尖端扭转型室性心动过速发作中,6次为电击除颤转复(20.7%),其余自行转复;8例应用异丙肾上腺素静脉滴注使心率维持在90～110/min,随后对其中3例植入临时起博器,给予90～110次/min的频率持续右心室心尖部起博,同时去除诱发因素,如纠正低钾血症,补镁,停用胺碘酮及其他引起Q-T间期延长的药物。结论获得性长Q-T间期综合征为心源性晕厥愿因之一,常诱发尖端扭转型室性心动过速,补钾、镁为重要错施。Objective To analyse the acquiredr of longQ-T internal syndrome.Methods The clinical data of 90 cases（60females and 30 males） with acquired of long Q-T internal syndrome were analysed in retrospect.Results Attack siuns tachycards 1 ～ 7 times/person,3.2 times in average,29 times in total.In which,6 times for defibrillation transfer back（20.7%）,the others transfer back by themselves.And 8 cases of application of isoproterenol infusion to maintain heart rate at 90 ～ 110times.Subs、quently,temporary pacemaker was implanted in the three cases of them to give them 90 ～ 110 times/min frequency continuos right ventricular apacial pacing.At the same time,remove the trigger factors,such as low solution that corrects hypokalemia,magnesium,disable amiadarone,and other drugs causingQ-Tinternal prolongation.Conclusion Acquired long Q-T internal prolongation.Conclusion Acquired long Q-T internal syndrome is one of the causes of cardiac syncope,often induced torsades sinus tachycardia,so replenishment of potarssium,magnesium is an important therapeutic measure.

关 键 词：获得性 长Q-T间期 综合征 

分 类 号：R541.7[医药卫生—心血管疾病]