力竭运动对大鼠血浆PGI_2、TXA_2与心肌ACP、MDA和β-GLU的影响研究  

The Study on Big Mouse Blood Plasma PGI2,TXA2 and Cardiac Muscle ACP,MDA,β-GLU after Sports Fatigue

作　　者：崔荣荣[1] 

机构地区：[1]温州医学院体育科学学院,浙江温州325035

出　　处：《吉林体育学院学报》2011年第1期76-79,共4页Journal of Jilin Sport University

摘　　要：分析大鼠力竭运动后血浆TXB2、6-keto-PGF1a变化与心肌ACP、MDA和β-GLU变化的相关性并探讨其机制,为预防运动性心肌损伤提供理论依据。结果显示:力竭运动即刻,血浆TXB2显著升高(P<0.01),心肌MDA、ACP、β-GLU变化与血浆TXB2、T/P显著正相关(P<0.05)。结论:血浆TXA2、PGI2平衡失衡可能是导致大鼠力竭运动后心肌损伤的重要原因。Studing correlation between the rat plasma TXB2,6-Keto-PGF1a and myocardial ACP,MDA and β-Glu after exercise and discuss its mechanisms,in order to provide a theory for preventing myocardial injury.The results：immediately,plasma TXB2 was significantly increased（P0.01）,while myocardial MDA,ACP,β-Glu and plasma TXB2,T/P were correlation（P0.05）.Conclusion：Plasma TXA2,PGI2 were imbalance might be the important reason what caused rats cardiac muscle damage after exercise.

关 键 词：力竭运动 心肌缺血 心肌损伤 

分 类 号：G804.2[文化科学—运动人体科学]