急性一氧化碳中毒后迟发性脑病发病机制的研究进展  

Progress on the Pathogenesis of Delayed Encephalopathy after Acute Carbon Monoxide Poisoning

作　　者：曲鹏飞[1] 杨冰[1] 杨晓蕾[1] 辛全兵[1] 武虓[1] 吕艳朋[1] 李杰[1] 

机构地区：[1]山东大学公共卫生学院劳动卫生与环境卫生学研究所,山东省济南市250012

出　　处：《职业与健康》2011年第19期2251-2254,共4页Occupation and Health

摘　　要：近年来,急性一氧化碳中毒后迟发性脑病(DEACMP)的发病率居高不下,国内为10%～30%,国外为0.8%～43%。然而,到目前为止其发病机制尚不清楚。作者总结分析近年来国内外相关研究,归纳DEACMP的几种发病机制,并提出其发病可能是多种机制协同作用的结果,以期为今后DEACMP的研究和预防治疗提供理论指导。In recent years,the incidence of Delayed Encephalopathy after Acute Carbon Monoxide Poisoning（DEACMP） remains high,which are 10%~30% in China,and 0.8%~43% in foreign countries.So far,its pathogenesis is still not clear.This article summarized and analyzed the related researches at home and abroad,concluded several pathogenesis of DEACMP,and suggested that the reason of DEACMP should be the synergism of various mechanisms,so as to provide the theoretical guidance for the research and prevention of DEACMP.

关 键 词：一氧化碳中毒 迟发性脑病 发病机制 

分 类 号：R595.1[医药卫生—内科学]