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作 者:赵林双[1] 向光大[1] 浦金辉[1] 侯洁[1] 乐岭[1] 孙慧伶[1] 谭学莹[1] 廖玉华[2] 王敏[2]
机构地区:[1]广州军区武汉总医院内分泌科,湖北430070 [2]华中科技大学同济医学院协和医院心内科
出 处:《中华内分泌代谢杂志》2011年第10期833-835,共3页Chinese Journal of Endocrinology and Metabolism
基 金:湖北省自然科学基金项目(2002AB116)
摘 要:观察371例糖尿病肾病患者、107例2型糖尿病患者和47名对照者血清β1肾上腺素能(B1)和血管紧张素Ⅱ1型(AT1)受体自身抗体阳性率与血清半胱氨酸蛋白酶抑素c(Cys—C)的关系。结果显示糖尿病肾病患者B1和AT1受体自身抗体阳性率明显高于2型糖尿病患者,糖尿病肾病患者中Cys—c异常者B1和AT1受体抗体阳性率明显高于Cys-C正常者。提示Cys—C异常与肾损害有关,更进一步表明糖尿病肾病与AT1和B1受体自身抗体有关。To observe the relationship between positive rate of β1-adrenergic and AT1 receptors autoantibodies with serum concentration of cystatin C in 371 patients with diabetic nephropathy patients, 107 patients with type 2 diabetes, and 47 subjects as healthy control. In patients with diabetic nephropathy, the positive rates of the β1 and AT1 receptors autoantibodies were significantly higher than those in patients with type 2 diabetes and normal controls. The titers of β1 and AT1 receptors autoantibodies in diabetic nephropathy patients with abnormal cystatin C were significantly higher than those with normal cystatin C concentration. These findings suggested that β1 and AT1 receptors autoantibodie may play important roles in the pathogenesis of diabetic neDhroDathv.
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