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作 者:梁维斌[1]
机构地区:[1]广西柳州市人民医院麻醉科
出 处:《华夏医学》1995年第1期1-2,共2页Acta Medicinae Sinica
摘 要:本组观察了22例全麻病人诱导插管期间SpO_2和PEtCO_2的变化。按插管无通气时限的不同分为A组(1min)和B组(2min)。通过面罩吸氧和手法人工通气,A、B两组病人SpO_2分别由96±0.45%和96±0.20%升至98±0.28%和98±0.16%,PEtCO_2分别由4.54±0.63kPa和4.63±0.40kPa降至3.41±0.60kPa和3.58±0.47kPa,前后比较有明显差异,两组间无差异。插管后A、B两组SpO_2均维持在98%以上,PEtCO_2则分别升到4.78±0.55kPa和4.93±0.58kPa,与插管前比较有显著差异,虽仍在正常范围内,但已呈明显上升趋势。提示了在插管无通气状态下1~2min内,缺氧不是主要的危险,而二氧化碳蓄积所带来的危害值得警惕和避免。The Changes of SpO_2 and PEtCO_2 during induction of anesthesia and intubation in 22 cases were observed 2 guoups were divided acording to the time limit of nonventilation: guoup A(1 minute)and guoup B(2 minutes). When the patients absorbed oxygen by mask and received manual artifieial)ventilation. In group A, SpO_2 increased from 96±0. 455 to 98±0. 28%and PEtCO_2 decreased from 4.45±0. 63kPa to 3. 58±0. 47kPa. In group B, SpO_2 gone up from 96 ±0. 40kPa to 3. 58 ±0. 47kPa. There was prominent significance compared pre-intubation with post-intubation, but there was no significance between group A and group B.After intubation, SpO_2 was kept above 90%in 2 guoups, PEtCO_2 increased to 4.78 ±0.55kPa in group A and to 4. 93 ± 0.58kPA in group B. It showed that hypoxemia is not main danger during intubation with non-ventilation for 1 minute or 2 minutes, but the accumulation of CO_2 should be warned and prevented.
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