褪黑素对慢性间歇性缺氧性心肌cTnT及Ca^(2+)-ATP酶的影响  

The Effect of Melatonin on the cTnT and Ca^(2+)-ATPase of Chronic Intermittent Hypoxia Cardiac Muscular Tissue

作　　者：凌月福[1] 张耀庭[1] 

机构地区：[1]桂林医学院附属医院耳鼻咽喉头颈外科,广西桂林541001

出　　处：《临床医学工程》2013年第3期383-384,共2页Clinical Medicine & Engineering

摘　　要：褪黑素(melatonin,MLT)是由松果体分泌的一种重要的激素。作为动物体内普遍存在的MLT具有多种生物学活性,如:提高睡眠质量、增加机体免疫力、抗氧化作用等。本文阐述MLT生物学作用及其对慢性间歇性缺氧性损伤心肌cTnT及Ca^(2+)-ATP酶的影响。Melatonin （melatonin, MLT） is one of the most important hormones secreted by the pineal gland. As the prevalence hormone inside of animal body, MLT has many biological activities, such as： improve sleep quality, enhancing immunity, anti-oxidation and so on. This article make a summary of MLT biological functions and effect of melatonin on the cTnT and Ca2＋ - ATPase of chronic intermittent hypoxia cardiac muscular tissues.

关 键 词：褪黑素 间歇性缺氧 CTNT CA^2+-ATP酶 

分 类 号：R335.1[医药卫生—人体生理学]