甲状腺功能亢进合并低血钾型周期性麻痹16例临床分析  

The thyroid gland function to clinical analysis of 16 cases of hyperthyroidism complicated with hypokalemic periodic paralysis

作　　者：王赞婷 

机构地区：[1]内蒙古牙克石市乌尔其汗医院,内蒙古 牙克石122159

出　　处：《世界最新医学信息文摘》2013年第25期38-38,31,共2页World Latest Medicine Information Electronic Version

摘　　要：目的：探讨甲状腺机能亢进（甲亢）合并低血钾型周期性麻痹的可能发病机制，临床特点及治疗方法。方法回顾性分析16例甲状腺功能亢进合并低血钾周期性麻痹的临床资料及诊治情况。结果16例病人均出现对称下肢瘫痪，重者合并双上肢瘫痪，发病时查血钾均低于正常。16例病人甲状腺功能测定显示 t3、t4均升高，tsH 降低。结论细胞免疫功能紊乱及离子通道异常及低血钾，可能是该病的发病机制。治疗时主要持续补钾，限制镁的摄入，抗甲状腺功能亢进是控制低钾麻痹症状多发的关键。Objective of hyperthyroidism （Graves） possible pathogenesis with hypokalemic periodic paralysis, clinical characteristics and treatment methods.Methods to review the clinical data and treatment analysis of 16 cases of hyperthyroidism complicated with hypokalemic periodic paralysis.Results 16 patients appeared symmetrical paralysis of the lower limbs, or with double upper limbs paralysis, the onset chaxuejia were lower than the normal. in 16 patients with thyroid function test showed that t3, t4 increased, tsH decreased.Conclusion the cellular immune function disorder and ion channel abnormalities and low blood potassium, may be the pathogenesis of this disease. treatment mainly for potassium, magnesium intake restriction, anti hyperthyroidism is the key to control the low potassium paralysis symptoms of multiple.

关 键 词：甲状腺功能亢进 低血钾 周期性麻痹 

分 类 号：R581.1[医药卫生—内分泌]