植物盐胁迫相关信号转导机制的研究  

Study on Signal Transduction Mechanism of Plant Salt Tolerance

作　　者：夏金婵[1] 张小莉[1] 

机构地区：[1]河南中医学院基础医学院,河南郑州450008

出　　处：《安徽农业科学》2014年第34期12023-12027,12030,共6页Journal of Anhui Agricultural Sciences

基　　金：河南中医学院博士基金(BSJJ2010-35);河南省科技攻关项目(112102310706)

摘　　要：植物的抗盐反应是一个复杂的过程,受许多基因的调控,外界的高Na+通过IP3诱导胞内Ca2+的升高,SOS3接收Ca2+信号,激活SOS2的激酶活性,SOS2通过调节位于质膜和液泡膜上的Na+/H+反向转运体把Na+运到体外或液泡中,ABA、ROS、At HK1、MAPK级联反应和LEA也参与盐胁迫造成的渗透胁迫和损伤的反应过程,但是要通过生物学手段利用盐信号传递过程中一些成分提高作物的抗盐能力,还需要对植物盐胁迫相关信号转导机制进行更加深入的研究。Salt related gene regulation network is a complex process,which includes many genes.Plant cells can sense high Na^＋ concentration,then increase cytosolic IP3 and Ca^2.Ca^2 signal was perceived by SOS3,which in turn activates the SOS2 kinase.The activated SOS2 kinase regulates sodium efflux and sequesters sodium into the vacuole by Na^＋/H^＋ antiporter,which express in plasma membrane and tonoplast.ABA,ROS,AtHK1,MAPK cascades and LEA are also involved in osmotic homeostasis and stress damage by salt stress.The signaling pathway component will be required to further understand to use in crop improvement.

关 键 词：植物 盐胁迫 SOS 转运体 

分 类 号：S432.31[农业科学—植物病理学]