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机构地区:[1]长春大学校医院,长春130022 [2]长春通源医院内科,长春130012
出 处:《长春大学学报》2014年第12期1690-1691,1695,共3页Journal of Changchun University
摘 要:选取44例COPD动脉血气为Ⅱ型呼吸衰竭患者,其中重度缺氧29例,中度缺氧15例。44例均为复合性酸碱失衡,呼吸型三重酸碱失衡10例。临床表现:手指静止性震颤39例,扑翼样震颤24例,呈"齿轮状"抵抗18例,头部颤动6例。对11例病情严重影响正常生活者给予美多巴试验性治疗5-7天治愈。得出:COPD低氧血症,高碳酸血症及酸碱失衡并发锥体外路系损害早期是可逆性的,积极治疗呼吸衰竭、纠正低氧血症,可防止并发锥体外路系统损害。44 cases of patients of respiratory failure from COPD with ]I type arterial blood gas are selected, among whom, 29 cases with severe anoxia and 15 cases with moderate anoxia. All of the 44 cases have compound acid-base unbalance, 10 cases with respiratory type triple acid-base unbalance disorders. Clinical symptoms:there are 39 cases with finger static tremor,24 cases with flapping tremor, 18 cases with gear-shaped resistance and 6 cases with head tremor. Madopar experiential treatment for 5 -7 days is given to 11 serious patients whose disease has affected their normal life. Conclusion: It is reversible for patients with early COPD hypoxemia, hypercapnia, acid-base unbalance and extrapyramidal system damage. The active treatment of respiratory failure and hypoxemia can prevent extrapyramidal system damage.
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