重症手足口病并发神经源性肺水肿的机制  

Pathogenesis of severe hand-food-mouth disease complicated with neurogenic pulmonary edema

作　　者：李伶芝 韦翊[1] 

机构地区：[1]温州医科大学附属育英儿童医院儿科,325027

出　　处：《国际儿科学杂志》2015年第1期55-58,共4页International Journal of Pediatrics

基　　金：浙江省卫生厅科技计划项目（2012KY159）

摘　　要：重症手足口病可并发神经源性肺水肿，严重者导致死亡。肺水肿的发生可能与中枢神经系统损伤后神经因素、体液因素、生物活性因子等多方面改变有关。研究重症手足口病并发神经源性肺水肿的发病机制从而采取积极有效的措施及时阻断并发症的发生具有重要意义。Severe handfoodmouth disease can be accompanied by neurogenic pulmonary edema, which can rapidly lead to death. The pathogenesis of neurogenic pulmonary edema may be related to the comprehensive influences on neural factors, humoral factors and multiple bioactive substances after the central nervous system is damaged. It is of great impotance to investingate the pathogenesis of neurogenic pulmonary edema, which facili tates timely treatment and prevention of the complications.

关 键 词：重症手足口病 肠道病毒71型 神经源性肺水肿 发病机制 

分 类 号：R725.1[医药卫生—儿科]