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作 者:Fang Xu Yu Ti Cheng Paul Kapos Yan Huang Xin Li
机构地区:[1]Michael Smith Laboratories, University of British Columbia, Vancouver, BC V6T 1Z4, Canada [2]Department of Botany, University of British Columbia, Vancouver, BC V6T 1Z4, Canada
出 处:《Molecular Plant》2014年第12期1801-1804,共4页分子植物(英文版)
摘 要:Dear Editor, Upon recognition of pathogen effectors, plant resist- ance (R) proteins trigger strong defense responses that restrict the growth and spread of pathogens. Most R pro- teins are nucleotide-binding (NB) and leucine-rich repeat (LRR) domain-containing proteins (NLRs) sharing struc- tural similarity with animal NOD-like receptors (Dodds and Rathjen, 2010). SNC1 (Suppressor of nprl, constitutive 1) is an Arabidopsis Toll/interleukin-1 Receptor (TIR)-type NLR that was originally identified through a gain-of-function autoimmune mutant, sncl, from a forward genetic screen.Dear Editor, Upon recognition of pathogen effectors, plant resist- ance (R) proteins trigger strong defense responses that restrict the growth and spread of pathogens. Most R pro- teins are nucleotide-binding (NB) and leucine-rich repeat (LRR) domain-containing proteins (NLRs) sharing struc- tural similarity with animal NOD-like receptors (Dodds and Rathjen, 2010). SNC1 (Suppressor of nprl, constitutive 1) is an Arabidopsis Toll/interleukin-1 Receptor (TIR)-type NLR that was originally identified through a gain-of-function autoimmune mutant, sncl, from a forward genetic screen.
分 类 号:Q243[生物学—细胞生物学] TS252.1[轻工技术与工程—农产品加工及贮藏工程]
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