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出 处:《Molecular Plant》2015年第5期671-673,共3页分子植物(英文版)
摘 要:Recent reports demonstrate a role of calmodulin (CaM)-binding DNA-binding proteins (transcription factors [TFs]) as repressors of the plant immune response through their interactions with promoters of a number of target genes. However, the role of Ca2+ in gene repression has not been tested. Thus, to infer the function of such TFs as repressors of transcription prior to path- ogen attack raises questions about the role of Ca2+ in this tran- scription repression and subsequently in de-repression upon pathogen attack. Here we discuss open questions and possible mechanisms.Recent reports demonstrate a role of calmodulin (CaM)-binding DNA-binding proteins (transcription factors [TFs]) as repressors of the plant immune response through their interactions with promoters of a number of target genes. However, the role of Ca2+ in gene repression has not been tested. Thus, to infer the function of such TFs as repressors of transcription prior to path- ogen attack raises questions about the role of Ca2+ in this tran- scription repression and subsequently in de-repression upon pathogen attack. Here we discuss open questions and possible mechanisms.
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