Receptor-mediated inhibitory mechanisms and the regulation of platelet function  

作　　者：CHENG ZhiPeng LIU JunLing 

机构地区：[1]Department of Biochemistry and Molecular Cell Biology, Shanghai Jiao Tong University School of Medicine [2]Institute of Hematology, Union Hospital, Tongji Medical College, Huazhong University of Science and Technology

出　　处：《Science China(Life Sciences)》2015年第12期1299-1301,共3页中国科学（生命科学英文版）

基　　金：supported by the National Natural Science Foundation of China(91439115);the National Basic Research Program of China(NO2012CB518000)

摘　　要：Platelets are derived from megakaryocytes;they circulate in mammalian blood vessels and play essential roles in hemostasis and thrombosis.When platelets encounter injury,they respond by triggering the formation of a blood clot to prevent blood loss(hemostasis).While this is vital for survival,Platelets are derived from megakaryocytes;they circulate in mammalian blood vessels and play essential roles in hemostasis and thrombosis.When platelets encounter injury,they respond by triggering the formation of a blood clot to prevent blood loss（hemostasis）.While this is vital for survival,

关 键 词：vital triggering Receptor encounter respond aggregation downstream prostaglandin PPAR thrombin 

分 类 号：R329.2[医药卫生—人体解剖和组织胚胎学]