植物盐胁迫的信号调控机制  

Signal Regulation Mechanism of Plant Salt Stress

作　　者：秦晓惠[1] 段志坤 Qin Xiaohui;Duan Zhikun(Key Laboratory of Plant Stress Biology,State Key Laboratory of Cotton Biology,Department of Biology,Henan University,Kaifeng,475004)

机构地区：[1]河南大学生命科学学院,棉花生物学国家重点实验室,植物逆境生物学教育部重点实验室

出　　处：《基因组学与应用生物学》2019年第8期3706-3713,共8页Genomics and Applied Biology

摘　　要：盐胁迫会引起渗透胁迫、离子毒害和次级胁迫,极大危害到植物的生存。之前的研究中,借助于分子生物学和遗传学手段,已经鉴定出多个基因参与到盐胁迫信号通路的调控,其中SOS途径是主要的植物抗盐调节机制。氨基酸、甜菜碱、可溶性糖等渗透调节物质,脯氨酸、多胺等信号小分子,SOD、CAT等抗氧化酶,HKT等离子通道蛋白均参与到了植物对盐胁迫的响应。本研究总结了盐胁迫条件下植物调控离子平衡、氧化胁迫、生长发育的最新研究进展,为之后的研究提供依据。Salt stress can cause osmotic stress, ion toxicity, and secondary tress, which are greatly harmful to crops. Many genes have been identified to involved in salt stress signal pathway by molecular biology and genetics in previous studies. Osmotic adjustment substance(e.g. amino acid, glycine betaine, soluble sugars), signaling molecule(e.g. proline, polyamine), antioxidase(e.g. SOD, CAT), ion channel(e.g., HKT) proteins all are allinvolved in salt stress regulation pathway. Recent progress of regulation of ion homeostasis by plants under salt stress,oxidative stress, growth and development under salt stress are summaried in this reviews as to provide a basis for subsequent research.

关 键 词：盐胁迫 SOS [Ca2+]cyt 离子平衡 渗透胁迫 ROS 

分 类 号：Q94[生物学—植物学]