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作 者:高佩虹 邵硕[1] 郑宁[1] 刘文娟[1] 赵振宇[2] 王光彬[3] GAO Peihong;SHAO Shuo;ZHENG Ning;LIU Wenjuan;ZHAO Zhenyu;WANG Guangbin(Department of Radiology, Jining First People’s Hospital, Jining 272000, P.R.China;Department of Neurology, Linyi People’s Hospital, Linyi 276000, P.R.China;Shandong Medical Imaging Research Institute, Affiliated to Shandong University, Jinan 250021, P.R.China)
机构地区:[1]山东省济宁市第一人民医院放射科,山东济宁272000 [2]山东省临沂市人民医院神经内科,山东临沂276000 [3]山东大学附属山东省医学影像学研究所,山东济南250021
出 处:《医学影像学杂志》2020年第8期1337-1340,共4页Journal of Medical Imaging
摘 要:目的探讨急性脑梗死后胼胝体压部可逆行性病变的发生机制。方法收集4例急性脑梗死后胼胝体压部可逆行性病变患者行MRI检查,结合文献进行分析。结果急性脑梗死后1~2周内胼胝体压部出现DWI呈高信号,ADC低信号的新发病变,临床未见与新发病变相关症状,1~2个月后胼胝体压部DWI高信号病变消失。结论急性脑梗死后可逆性胼胝体病变是华勒氏变性早期改变,其应与脑梗死、可逆性胼胝体压部病变综合征及原发性胼胝体变性等疾病鉴别。Objective To study the mechanism underlying transient lesions in the splenium of the corpus callosum(SCC)secondary to acute ischemic stroke.Methods 4 cases who showed transient lesions in the SCC secondary to acute ischemic stroke were described and analyzed according to the clinical and MR features,combined with the literature.Results Both patients showed a fresh lesion in the SCC on magnetic resonance imaging(MRI)1~2 weeks after the onset of stroke,with a hyperintense lesion on diffusion-weighted imaging and decreased apparent diffusion coefficient values,with no symptoms related to the lesion.In both cases,the lesion disappeared on MRI 1~2 months later.Conclusion Transient lesions in the SCC secondary to acute ischemic stroke are early wallerian degeneration after cerebral infarction.Clinicians should note that transient SCC lesions can occur following acute ischemic stroke and avoid misdiagnosing them.
分 类 号:R445.2[医药卫生—影像医学与核医学] R743[医药卫生—诊断学]
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