动脉扩张性血管瘤与阻塞性睡眠呼吸暂停低通气综合征的关系研究进展  

Research Progress on the Relationship between Arterial Dilated Hemangioma and Obstructive Sleep Apnea Hypopnea Syndrome

作　　者：李美美[1] LI Meimei(The Second Affiliated Hospital of Fujian Medical University,Quanzhou 362000,China)

机构地区：[1]福建医科大学附属第二医院,福建泉州362000

出　　处：《中外医学研究》2020年第35期186-188,共3页CHINESE AND FOREIGN MEDICAL RESEARCH

基　　金：福建省自然科学基金项目(2016J01441)。

摘　　要：阻塞性睡眠呼吸暂停低通气综合征(OSAHS)主要临床表现为睡眠过程中反复性出现上气道阻塞、塌陷,致使呼吸中断或/和出现不同程度的低通气,引起睡眠结构紊乱、高碳酸血症,导致患者出现打鼾、白天嗜睡、睡眠结构紊乱等症状,损害患者的神经系统、代谢系统、消化系统、泌尿生殖系统、内分泌系统,导致患者的免疫功能、性功能、代谢功能出现异常。临床实践可知,OSAHS是导致人们发生高血压、心脑血管疾病(脑卒中、冠心病等)、肺源性心脏病的一项独立危险因素,OSAHS将会影响人体胸腔内压力的波动情况,导致机体的血压增高,引起动脉粥样硬化(AS),严重损害患者的机体,而这些症状的发生都会引起动脉扩张性血管瘤,或者加速动脉扩张性血管瘤的进展。The main clinical manifestations of obstructive sleep apnea hypopnea syndrome (OSAHS) are repeated upper airway obstruction and collapse during sleep,resulting in respiratory interruption or/and varying degrees of hypopnea,causing sleep structure disorders,hypercapnia causes the patient to have symptoms such as snoring,daytime sleepiness,sleep structure disturbance,damage to the patients’ nervous system,metabolic system,digestive system,genitourinary system,and endocrine system,resulting in the patients’ immune function,sexual function,and metabolic function.abnormal.According to clinical practice,OSAHS is an independent risk factor that causes people to develop hypertension,cardiovascular and cerebrovascular diseases (stroke,coronary heart disease,etc.),and pulmonary heart disease.OSAHS will affect the fluctuation of pressure in the human chest cavity,leading to the body’s blood pressure increases,causing atherosclerosis (AS),which severely damages the patients’ body,and the occurrence of these symptoms can cause arterial dilated hemangioma,or accelerate the progress of arterial dilated hemangioma.

关 键 词：动脉扩张性血管瘤 阻塞性睡眠呼吸暂停低通气综合征 影响机制 关系 

分 类 号：R766[医药卫生—耳鼻咽喉科]