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作 者:冯超[1] 冯津萍[1] 陈树涛[1] 郭玉军[1] Feng Chao;Feng Jinping;Chen Shutao;Guo Yujun(Department of Cardiology,Tianjin Chest Hospital,Tianjin 300222,China)
出 处:《中华心力衰竭和心肌病杂志(中英文)》2021年第2期118-122,共5页Chinese Journal of Heart Failure and Cardiomyopathy
摘 要:甲状旁腺激素(PTH)主要调节钙、磷代谢,甲状旁腺功能减退会导致血钙降低、血磷浓度增高。长期而严重的低钙血症可能因心肌收缩力受损而引起继发性扩张型心肌病,出现充血性心力衰竭,称为甲状旁腺功能减退性心肌病。本文报道一例37岁男性甲状腺全切术后甲状腺及甲状旁腺功能减退症患者,因射血分数减低的心力衰竭入院,经标准的抗心力衰竭药物治疗及积极补充钙剂、活性维生素D等治疗后,患者的心脏结构和功能显著改善。Parathyroid hormone(PTH)mainly regulates the metabolism of calcium and phosphorus,and hypoparathyroidism can cause decrease in serum calcium and increase in serum phosphorus concentration.Chronic and severe hypocalcemia may lead to secondary dilated cardiomyopathy due to impaired myocardial contractiity,resulting in congestive heart failure,known as hypoparathyroidism cardiomyopathy.In this paper,we reported a 37-year-old male patient with hypoparathyroidism after total thyroidectomy who was admitted to hospital with heart failure with reduced ejection fraction.He was treated with standard anti-heart failure medications,plus active supplementation of calcium and active vitamin D and significant improvement in his heart structure and function occurred during follow-up.
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