氧化应激与糖尿病周围神经病变的机制研究进展  

Research Progress on the Mechanism of Oxidative Stress and Diabetic Peripheral Neuropathy

作　　者：吕培然 裴建[2] 高正 沈雪勇[1] 邓海平[1] LYU Peiran;PEI Jian;GAO Zheng;SHEN Xueyong;DENG Haiping(Shanghai University of Traditional Chinese Medicine,Shanghai 201203,China;不详)

机构地区：[1]上海中医药大学,上海201203 [2]上海中医药大学附属龙华医院

出　　处：《中国医学创新》2022年第2期185-188,共4页Medical Innovation of China

基　　金：上海市卫健委海派中医传承工程(ZY(2018-2020)-CCCX-1006)。

摘　　要：糖尿病周围神经病变是糖尿病主要并发症之一,以神经受损为主要病理改变,其发病机制与氧化应激反应关系密切。文章综合近年来的研究成果,总结阐述氧化应激参与糖尿病周围神经病变的作用机制。Diabetic peripheral neuropathy is one of the main complications of diabetes,with nerve damage as the main pathological change,and its pathogenesis is closely related to oxidative stress response.Based on the research results of recent years,the mechanism of oxidative stress in diabetic peripheral neuropathy is summarized.

关 键 词：氧化应激 糖尿病周围神经病变 发病机制 

分 类 号：R587.2[医药卫生—内分泌]