甲乙型流感对患儿肌酸激酶不同影响病例报道1例  

A case report of different effects of influenza A and B on creatine kinase in children

在线阅读下载全文

作  者:吴亚楠 袁红梅[1] 焦素敏[1] WU Yanan;YUAN Hongmei;JIAO Sumin(Department of Pediatrics,Beijing Jingmei Group General Hospital,Beijing 102300,China)

机构地区:[1]北京京煤集团总医院儿科,北京102300

出  处:《中国医药科学》2022年第12期194-196,共3页China Medicine And Pharmacy

摘  要:流行性感冒是一种由流感病毒引起的急性呼吸道传染病,潜伏期短,经呼吸道飞沫传播,传播速度快,流行呈一定的季节性,每年10月我国各地陆续进入流感流行季节,虽然流感病毒感染大部分为自限性,但是仍有少数进展为重症。儿童是流感病毒感染的高危人群。本文报道1例患儿4个月内,先后感染甲型流感和乙型流感,感染后肌酸激酶均出现不同程度升高,乙型流感病毒感染后肌酸激酶升高近20倍,经积极治疗后肌酸激酶下降至正常,且无后遗症,故诊断为重症流感、儿童急性良性肌炎。借此病例以提示临床医生对不同流感引起肌酸激酶不同变化的认识,复习重症流感的诊断标准,警惕重症流感及其并发症的出现,为临床工作提供借鉴。Influenza is an acute respiratory infectious disease caused by influenza virus,which has a short incubation period and spreads quickly through respiratory droplets,and the epidemic is seasonal.Every October,all parts of our country enter the influenza epidemic season one after another.Although most of the infection with influenza virus is self-limited.there are still a few cases of severe influenza.Children are at high risk of influenza virus infection.This paper reports a case of a child who was successively infected with influenza A and influenza B within four months.the creatine kinase increased in varying degrees after infection,however,the creatine kinase increased nearly 20 times after infection with influenza B virus.after active treatment,creatine kinase decreased to normal,and there were no sequelae,so it was diagnosed as severe influenza and acute benign myositis.This case is used to suggest clinicians’understanding of different changes of creatine kinase caused by different influenza,review the diagnostic criteria of severe influenza,and guard against the emergence of severe influenza and its complications,so as to provide ref erence for clinical work.

关 键 词:甲型流感 乙型流感 肌酸激酶 儿童 急性良性肌炎 

分 类 号:R511.7[医药卫生—内科学]

 

参考文献:

正在载入数据...

 

二级参考文献:

正在载入数据...

 

耦合文献:

正在载入数据...

 

引证文献:

正在载入数据...

 

二级引证文献:

正在载入数据...

 

同被引文献:

正在载入数据...

 

相关期刊文献:

正在载入数据...

相关的主题
相关的作者对象
相关的机构对象