心搏骤停后重要器官损伤病理生理机制及相互作用研究进展  

Advances on pathophysiology and interaction of heart,brain and kidney injury after cardiac arrest

作　　者：唐婷婷(综述) 廉应涛 余追(审校)[1] TANG Ting-tingreviewing;LIAN Ying-tao;YU Zhuichecking(Department of Critical Care Medicine,Renmin Hospital of Wuhan University,Wuhan 430060,Hubei,China;Department of Anesthesiology,Renmin Hospital of Wuhan University,Wuhan 430060,Hubei,China)

机构地区：[1]武汉大学人民医院重症医学Ⅱ科,武汉430060 [2]武汉大学人民医院麻醉科,武汉430060

出　　处：《医学研究生学报》2022年第7期772-777,共6页Journal of Medical Postgraduates

基　　金：国家自然科学基金(81772039)。

摘　　要：心搏骤停自主循环恢复后会出现复苏后多器官功能不全。心、脑、肾是常见受损器官,而各器官损伤的病理生理过程复杂,涉及多种机制,损伤器官之间又可相互作用,进一步加重病情,降低存活率。但其相关机制尚未完全阐明,现对心搏骤停后心、脑、肾损伤的病理生理机制以及相互作用作一综述。Post-resuscitation multi-organ dysfunction can occur after return of spontaneous circulation following cardiac arrest.Heart,brain and kidney are commonly injured accompanied with complex pathophysiology and multiple mechanisms.And their interaction further exacerbates patients'condition and reduces the survival rate.Currently,the relevant mechanisms have not been fully clarified.This review summarized the pathophysiology and the interaction of heart,brain and kidney injury after cardiac arrest.

关 键 词：心搏骤停 心肌损伤 脑损伤 急性肾损伤 

分 类 号：R541.78[医药卫生—心血管疾病]