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作 者:Elaine Chow Juliana C.N.Chan 周怡君;陈重娥(Department of Medicine and Therapeutics,The Chinese University of Hong Kong,Prince of Wales Hospital,Hong Kong,China;Hong Kong Institute of Diabetes and Obesity,The Chinese University of Hong Kong,Prince of Wales Hospital,Hong Kong,China;Phase 1 Clinical Trial Centre,The Chinese University of Hong Kong,Prince of Wales Hospital,Hong Kong,China;Li Ka Shing Institute of Health Sciences,The Chinese University of Hong Kong,Prince of Wales Hospital,Hong Kong,China)
机构地区:[1]Department of Medicine and Therapeutics,The Chinese University of Hong Kong,Prince of Wales Hospital,Hong Kong,China [2]Hong Kong Institute of Diabetes and Obesity,The Chinese University of Hong Kong,Prince of Wales Hospital,Hong Kong,China [3]Phase 1 Clinical Trial Centre,The Chinese University of Hong Kong,Prince of Wales Hospital,Hong Kong,China [4]Li Ka Shing Institute of Health Sciences,The Chinese University of Hong Kong,Prince of Wales Hospital,Hong Kong,China
出 处:《Science Bulletin》2022年第23期2392-2394,共3页科学通报(英文版)
摘 要:Type 2 diabetes (T2D) is characterised by defects in both fasting and postprandial glucose (PPG) control. Many Asians including Chinese people with T2D have isolated high PPG at diagnosis without concomitant elevation in fasting plasma glucose (FPG). This might be due to differences in genetic architecture, low insulin secretion, and high carbohydrate diet in East Asians. Excursions in PPG contribute significantly to glycemic variability (GV).
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