王书臣从“气虚血损,毒络肺积”论治原发性气管支气管肺淀粉样变性  

Treatment of Wang Shuchen for Primary Tracheobronchopulmonary Amyloidosis from“Qi Deficiency and Blood Loss,Toxin Collaterals and Lung Accumulation”

作　　者：周瑞玲 王冰[2] 丛晓东[2] 张雯 张新宇 张琼[2] ZHOU Ruiling;WANG Bing;CONG Xiaodong;ZHANG Wen;ZHANG Xinyu;ZHANG Qiong(Beijing University of Chinese Medicine,Beijing 100029,China;Xiyuan Hospital of China Academy of Chinese Medical Sciences,Beijing 100091,China)

机构地区：[1]北京中医药大学,北京100029 [2]中国中医科学院西苑医院,北京100091

出　　处：《中国中医药信息杂志》2023年第7期174-177,共4页Chinese Journal of Information on Traditional Chinese Medicine

基　　金：国家自然科学基金(81673817);中国中医科学院科技创新工程(CI2021A01106、CI2021A01104);北京中医药薪火相传“3+3”项目-王书臣名医传承工作站(2019年)。

摘　　要：王书臣教授认为,原发性气管支气管肺淀粉样变性(PTBA)核心病机为本虚标实,以气虚血损为本,毒络肺积为标,其内涵为肺气络亏虚,血络受损,毒瘀胶结,损络成积。络以通为用,当益气活血解毒散结以通络消积,标本兼治,补中有通,补而不滞,寓通于补,通而不损。本文结合医案总结王老治疗PTBA经验,以期为中医相关治疗提供辨治思路。Professor Wang Shuchen believes that the core pathogenesis of primary tracheobronchopulmonary amyloidosis(PTBA)is excess in nature and deficiency in superficiality,with the deficiency of qi and blood loss as the root cause and toxin collaterals and lung accumulation as the symptoms,which internal connotation is deficiency of qi collaterals,blood collaterals damage,toxin and stagnation,and damage to collaterals to form accumulation.The collaterals are used to pass;therefore,tonifying qi,activating blood circulation,detoxification and resolving mass should be used to activate collaterals and expelling accumulation to simultaneously treat the symptoms and root cause,with tonifying in activating,tonifying without stagnation,integrating activating with tonifying,and activating without damage.This article summarized Wang’s experience in treating PTBA by combining medical cases,with a view to providing ideas for the identification and treatment of related TCM treatment.

关 键 词：名医经验 王书臣 原发性气管支气管肺淀粉样变性 气虚血损 毒络肺积 

分 类 号：R249[医药卫生—中医临床基础]