Commentary on“Synchronized activity of sensory neurons initiates cortical synchrony in a model of neuropathic pain”  

作　　者：Lorenzo Di Cesare Mannelli Carla Ghelardini 

机构地区：[1]Department of Neuroscience,Psychology,Drug Research and Child Health-Neurofarba-Section of Pharmacology and Toxicology,University of Florence

出　　处：《Neural Regeneration Research》2024年第4期728-728,共1页中国神经再生研究（英文版）

摘　　要：In pat i e nt s,a s well as in animal mod e l s,hypersensitivity to external stimuli(hyperalgesia and allodynia)or spontaneous pain is often the first,and the most disabling,symptom of neuropathy(Davis et al.,2020).The increased activity of sensitive neurons drives pain development,making ion channel modulation a fundamental target for current pharmacotherapy as well as one of the most investigated by the R&D departments of pharmaceutical companies(Bennett et al.,2019).

关 键 词：COMPANIES PAIN ENT 

分 类 号：R338[医药卫生—人体生理学]