Lonicera Japonica Caulis Ameliorates LPS and TNF-α-Induced HT-29 Cell Injury by Inhibiting the MAPK/ERK/JNK/p38 Pathway  

作　　者：Hanxiu Lu Wei Yan Xiao Yuan Yongbo Kang Zhongjian Liu Qiang Guo 

机构地区：[1]Faculty of Life science and Technology,Kunming University of Science and Technology,Kunming 650031,Yunnan,China [2]School of medicine,Kunming University of Science and Technology,Kunming 650031,Yunnan,China [3]Department of Gastroenterology,The First People’s Hospital of Yunnan Province,Kunming 650032,Yunnan,China [4]Department of Gastroenterology,The First People’s Hospital of Yuxi City,Yuxi 653100,Yunnan,China [5]Institute of Basic and Clinical Medicine,The First People’s Hospital of Yunnan Province,Kunming 650032,Yunnan,China

出　　处：《Biomedical and Environmental Sciences》2024年第7期805-810,共6页生物医学与环境科学（英文版）

基　　金：Yunnan Digestive Endoscopy Clinical Medical Center Foundation[2X2019-01-02]for Health Commission of Yunnan[2022LCZXKF-XH13,2022LCZXKF-XH15 and 2022LCZXKF-XH19];Fundamental Research Projects of Yunnan Province[202201AU070013]。

摘　　要：Inflammatory bowel disease(IBD)is characterized by chronic inflammation of the intestinal tract,and shows a high rate of recurrence.Clinicians have accumulated extensive experience in the treatment of IBD,with biologics and small molecule drugs having greatly expanded the therapeutic scope of IBD in recent years.Nonetheless,such treatments involve a high price and a single target of drug action.

关 键 词：drugs treatment P38 

分 类 号：R285[医药卫生—中药学]