创伤性脑损伤后的代谢变化和营养支持  

The metabolic response to traumatic brain injury and implications for nutritional support

作　　者：杭春华[1] 史继新[1] 

机构地区：[1]南京大学医学院临床学院南京军区南京总医院神经外科，江苏南京210002

出　　处：《肠外与肠内营养》2003年第4期232-235,共4页Parenteral & Enteral Nutrition

摘　　要：创伤性脑损伤后代谢变化包括高代谢、高分解代谢、高血糖、急性相反应和免疫功能抑制等。足够的营养支持和恰当的代谢调理对减轻继发性脑损害、减少感染发生率和改善预后有重要的临床意义 。The metabolic response to traumatic brain injury includes hypermetabolism, hypercatabolism, hyperglycemia, acute phase response and immunosuppression, which are mediated mainly by hypothalamus pituitary adrenal axis and cytokines. Sufficient nutritional support and appropriate metabolic intervention are believed clinically to play an important role on the mitigation of secondary brain damage, reduction of septic rate and improvement of patient′s outcomes.

关 键 词：创伤性脑损伤 代谢功能 营养支持 预后 急性相反应 免疫功能 

分 类 号：R651.15[医药卫生—外科学]