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作 者:孙校楼[1] 齐全[1] 邵秀芝[1] 苑晶慧[1] 程文增[1] 林福春[1] 高屋洁 滕盛启成 大友浩志[2] 高桥达也 田口喜雄 张德恒[3] 孙辉[3] 黄平[3] 白日星[3]
机构地区:[1]吉林省地方病第二防治研究所 [2]日本国东北大学医学部 [3]白求恩医科大学第三临床医院
出 处:《中国地方病防治》1992年第6期327-328,330,共3页Chinese Journal of Control of Endemic Diseases
摘 要:本文应用日本Atoka公司产浅表超声仪对缺碘地区长期供碘后和非缺碘地区甲状腺形态进行了各个切面的描记。结果是;居住在缺碘地区人群,甲状腺异常改变率占28.67%,而非缺碘地区为9.94%。二者差异非常显著,缺碘与非缺碘地区甲状腺改变异常率均女高于男。异常改变主要发生在>30岁年龄组;而<30岁年龄组则无差异。原因是从1958年后缺碘地区开始供应碘盐,机体缺碘状态已被纠正,而>30岁年龄组是供碘盐前出生并居住在病区,是因缺碘而造成的甲状腺损害,致病后虽供应了碘盐,然而甲状腺内部却残留了不同程度的病理损害,这些损害主要是形成腺瘤样甲状腺肿、结节性甲状腺肿、甲状腺钙化、甲状腺恶变及桥本氏病等。The morphology of thyroids in iodine deficiency areas after long-term iodine supply and that in non-iodine deficiency areas were studied by recording sections in different planes with superficial ultrasono scope. The results showed that the rate of abnormal changes in thyroids accounted for 23.6% in inhabitants of iodine deficiency areas while that in non-iodine deficiency areas, 9.94%, the difference being very significant. The rates of abnormal changes in female were higher than those in male in both the iodine and noniodine deficiency areas, showing abnormal changes mainly in age groups over 30 years, with no difference in age groups under 30.It was due to iodine supply to the iodine deficiency areas since 1958,while those in age groups over 30 were born before iodine supply to and lived in the diseased areas with thyroid lesions due to iodine deficiency. Although iodine has been supplied after having the disease, pathologic lesions of different degrees were remained in thyroids. They were responsible for the formation of adenomatoid goiter, nodular goiter, thyroid calcification, thyroid malignancies and Hashimoto's disease, etc.
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