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机构地区:[1]新汶矿业集团汶南矿医院,山东新汶271202
出 处:《实用医技杂志》2006年第8期1349-1350,共2页Journal of Practical Medical Techniques
摘 要:目的:探讨出血性脑卒中后BHS的临床特点和发病机制。方法:对52例出血性脑卒中合并BHS患者的临床资料进行回顾性分析。结果:BHS在出血性脑卒中患者中的发病率为36.36%,蛛网膜下腔出血发生BHS明显高于脑出血(P<0.05)。BHS的心电图主要表现为心肌缺血、心律失常和类心肌梗死。88.46%的患者于出血后1周内发生BHS。结论:BHS与出血性脑卒中的类型有关,其发病机制与出血性脑卒中导致植物神经失调有关。Objective To explore the clinical characteristics and pathogenesis mechanism of Brain-heart Syndrome after hemorrhagic brain stroke. Methods To give a retrospective analysis of clinical data in 52 patients with BHS after hemorrhagic brain stroke. Results The rate of pathogenesis in the patients with BHS in hemorrhagic brain stroke was 36.36%. BHS in subarachnoid hemorrhage was much higher than that in cerebral hemorrhage (P〈0. 05). The main presentation of electrocardiographic in BHS was myocardial ischemia dgsrhgthmias and like myocardial infarction 88. 46% patients had BHS after hemorrhage with in one week. Conclusions BHS relates to hemorrhagic stroke. Its pathogenesis mechanism may relate to hemorrhagic stroke causing antonomic imbalance.
分 类 号:R743[医药卫生—神经病学与精神病学]
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