外伤性腔隙性脑梗死的病理因素及CT相关分析  

Pathologic factors and CT manifestation of taumatic cerebral lacunar infarction

作　　者：张树宝[1] 颜广林[1] 张波[1] 贺小平[1] 

机构地区：[1]江苏省连云港市第一人民医院影像科,222002

出　　处：《蚌埠医学院学报》2010年第1期64-65,共2页Journal of Bengbu Medical College

摘　　要：目的:分析外伤性腔隙性脑梗死的病理因素与病变发生的关系。方法:使用全身螺旋CT扫描仪进行颅脑扫描,并分析其CT表现。结果:外伤性腔隙性脑梗死均发生在基底核内囊区,表现为斑片状、点片状类圆形低密度影,边缘欠清晰,其中双侧11例,单侧28例,合并有豆状核区钙化29例,钙化呈斑块状、斑点状及砂粒状。结论:基底核区钙化的存在是外伤性腔隙性脑梗死的主要病理基础。外伤所致脑组织的不均匀肿胀,引起基底核区钙化的移位,使血管受压,导致供血区域的脑组织梗死是基底核区外伤性腔隙性脑梗死的主要病理因素。Objective：To analyze the relationship between the pathologic factors of traumatic cerebral lacunar infarction（TCLI） and the occurrence of the disease.Methods：Spiral CT scan and brain scan were performed in 39 cases of TCLI,and the clinical manifestation and CT findings were analyzed.Results：All the TCLI occurred in the region of internal capsule,appearing as small sheet or small round shadow with unclear margin.The lesion was located in double hemisphere in 11 cases and in unilateral hemisphere in 28 cases.Twenty-nine cases were accompanied by lenticular nucleus calcification in speckle,dot and sand-like high density shadow.Conclusions：The calcification in the basal ganglia is the main pathologic basis of TCLI.The injured uneven swollen cerebral tissue can cause displacement of calcification in the basal ganglia and press the vessel,which result in infarction of blood-supply cerebral tissue,and is the main pathologic factor for basal ganglia TCLI.

关 键 词：脑梗死 脑损伤 病理 体层摄影术 X线计算机 

分 类 号：R743.33[医药卫生—神经病学与精神病学]