超声生物显微镜在青少年挫伤性近视发病机制的应用研究  

Study of the mechanism of traumatic myopia in teenagers using ultrasound bio-microscopy

作　　者：朱子诚[1] 柯根杰[1] 潘红飙[1] 

机构地区：[1]安徽医科大学附属省立医院眼科,合肥230001

出　　处：《山东大学耳鼻喉眼学报》2010年第1期54-56,60,共4页Journal of Otolaryngology and Ophthalmology of Shandong University

摘　　要：目的利用超声生物显微镜(UBM)初步探讨青少年挫伤性近视的发病机制。方法随机选取伤后表现为近视患者24例26眼,分别在急性期(伤后3d内)和恢复期(伤后2星期)测量屈光度、晶体厚度,UBM检测角膜厚度、前房轴深、小梁睫状突距离(TCPD)、A角、睫状突的高度(T值)。同时对20眼正常对照组进行2次上述测量,间隔15d。结果近视度随眼挫伤的好转而下降;晶体厚度、前房轴深、小梁睫状突距离(TCPD)、A角、睫状突的高度(T值)在急性期和恢复期差异均有统计学意义(P<0.05),而角膜厚度无明显变化(P>0.05)。结论睫状肌痉挛,前房变浅,晶体变凸是青少年挫伤性近视的共同机制。而睫状突肿胀、睫状体位置的改变是导致前房变浅、晶体变凸的原发因素。UBM可精确测定反映睫状突位置和高度的变化,对定量测量挫伤性近视的形态变化,具有重要的临床价值。Objective To approach the mechanisms of traumatic myopia using ultrasound biomicroscopy （UBM）. Methods 24 cases （26 eyes） with blunt eye trauma were included in this study. Refraction and lens thickness were measured respectively with an autorefractometer and A-scan ultrasoundgraphy in the acute（ 3 days after blunt eye injury） and convalescent stages（2 weeks after injury）. The cornea thickness（ CT）, central anterior chamber depth（ CACD）, trabecular ciliary process distance （TCPD）, A-angle （AA） and the T values （TV） were measured by ultrasound bio-microscopy （UBM）. 20 normal eyes as the control group were measured twice as mentioned above. Results The refraction, lens thickness, anterior chamber depth, TCPD, A-angle and T values had statistical difference between the acute and convalescent stages （P 〈 0.05 ） but cornea thickness had no change （ P 〉 0.05 ）. Conclusion The mechanisms in traumatic myopia are related with ciliary spasm, shallower anterior chamber and thickened lens. But primitive factors resuiting in these changes are diffuse edema of the ciliary body and anterior rotation of the entire ciliary body. UBM can measure pathological changes in traumatic myopia and has important clinical value.

关 键 词：超声生物显微镜 眼损伤 近视 发病机制 

分 类 号：R779.6[医药卫生—眼科]