斑点叉尾鮰细菌性败血症的组织病理研究  

Histopathologic Observation on Septicemia of Ictaclurus Punctatus

作　　者：戴振炎[1] 王荣华[1] 李权生 刘晓燕[1] 肖调义[1] 罗方兴[1] 陈冬香[1] 

机构地区：[1]湖南农业大学动物科学技术学院,中国长沙410128 [2]安化县移民局,中国安化413500

出　　处：《湖南师范大学自然科学学报》2012年第4期64-69,共6页Journal of Natural Science of Hunan Normal University

基　　金：湖南省科技厅基金资助项目(2010NK3025;2011NK3074)

摘　　要：对具有典型细菌性败血症症状的斑点叉尾鮰进行了系统的病理研究,组织性病变主要表现为:鳃小片上皮细胞增生,相互融合在一起呈板块状,呼吸上皮细胞肿胀变性;肠上皮细胞变性、坏死、脱落,固有膜和黏膜下层毛细血管扩张淤血;肝细胞肿胀,空泡变性,甚至发生坏死,肝细胞索结构排列紊乱;胃黏膜上皮细胞变性、坏死、脱落,固有膜毛细血管扩张充血,部分固有膜裸露;肾小管上皮细胞肿胀、变性、坏死,肾小球、肾间质出现水肿、出血现象;肌纤维明显肿胀断裂,出现团块状均质化变性,或发生不规则溶解.The pathology of Ictaclurus punctatus with the typical symptoms bacterial septicemia was studied. The results demonstrated that the main pathological changes of bacterial septicaemia in Ictaclurus punctatus were as follows： Gill lamellae showed proliferation and fusion as a plate, tumefaction and degeneration in the epithelial cells. Intestine showed degeneration, necrosis, loss in epithelial cells, expansion and congestion in capillary of lamina propria and submucosa. Liver ceils showed tumefaction, vacuolar degeneration and even necrosis, and hepatic cord arranged irregularly. The stomach showed degeneration, necrosis, loss in epithelial cells, expansion and congestion in capillary of lamina propria and part of the lamina pmpria exposed. The kidney showed swelling, degeneration, necrosis in renal tubular epithelial cell, atrophy and even necrosis, collapse in glomemlar, edema, hemorrhage in interstitial. The muscle fibers were swelled and fractured, with cloddy, homogenizing degeneration or irregular dissolution.

关 键 词：斑点叉尾鮰 组织病理学 细菌性败血症 

分 类 号：S943[农业科学—水产养殖]