急性一氧化碳中毒后迟发性脑病发病机制研究进展  

Progress of Pathogenesis of Delayed Encephalopathy after Acute Carbon Monoxide Poisoning

作　　者：陈炳达 李经伦[1] 

机构地区：[1]泸州医学院附属医院神经内科,四川省泸州市646000

出　　处：《实用心脑肺血管病杂志》2014年第4期1-4,共4页Practical Journal of Cardiac Cerebral Pneumal and Vascular Disease

摘　　要：急性一氧化碳中毒后迟发性脑病(DEACMP)的发病机制至今仍未完全清楚,多年来,国内外学者对其进行了大量研究,认为DEACMP的发生是多种因素综合作用的结果。本文对近年来DEACMP发病机制的研究进展进行综述,以期为DEACMP的研究和防治提供参考。The pathogenesis of delayed encephalopathy after acute carbon monoxide poisoning （DEACMP） has not been fully elucidated yet. Over the years, domestic and foreign scholars have conducted extensive research on it, they believe that DEACMP is the result of many faetors＇combined effect. This article summarizes the relevant research of DEACMP in recent years, in order to provide a reference to the research and prevention.

关 键 词：一氧化碳中毒 迟发性脑病 发病机制 

分 类 号：R743[医药卫生—神经病学与精神病学]